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Control of Tomato Spotted Wilt Virus in Floral Crops 1995 Proposal


DEVELOPMENT OF RESISTANCE


TO TOMATO SPOTTED WILT AND OTHER SIMILAR


VIRUSES IN FLORAL CROPS

Dr. James W. Moyer
North Carolina State University
1995

INTRODUCTION
This is a request for continuation of funding of a project whose primary
objective is

to introduce genes for resistance to tomato spotted wilt virus (TSWV) and
impatiens

necrotic spot virus (INSV; formerly TSWV-I) into selected floral crops.
The entire group

of tomato spotted wilt-like viruses are now known collectively as the Tospoviruses.
During

the past five years we have been investigating the Tospoviruses which infect
floral and other

crops. The American Floral Endowment has been a primary supporter of this
research. We

feel very fortunate to have been able to increase the level of understanding
to the point that

we are now ready to extend this research directly to the development of
resistant cultivars

and thus provide a more efficient means of control for these viruses.
Under previous funding from the American Floral Endowment we identified
the new

virus, INSV, as the predominant Tospovirus in greenhouse-grown floral crops
(14,15,19).

When our research began on spotted wilt, it was widely accepted that the
disease was caused

by a single virus (29, AFE symposium, Sanibel Island). Our research and
that of others in

Europe has now shown that there are a number of variants and at least two
distinct viruses

(4,5,7,10,14,15,26). We were the first to recognize the existence of the
second Tospovirus

(17). This virus was provisionally designated TSWV-I, but we have shown
that it is

sufficiently distinct to be a separate virus and named it impatiens necrotic
spot virus (INSV)

(16,17). Development of antisera for identification of INSV has allowed
for accurate

diagnosis of this virus in floral crops.
Our research on identification and characterization of Tospoviruses has
allowed us to

use genetic engineering techniques to develop resistance to these viruses.
We have isolated

the nucleocapsid (or “N”) gene from both viruses (16). Extensive research
with similar

genes from other viruses has documented the efficacy of nucleocapsid or
coat protein genes

in protecting plants against virus infection following transfer and expression
of these genes in

plants (1, 21). Although the mechanism involved in virus protection is
not understood,

extensive laboratory and field trials with crop plants transformed with
such genes has

documented the high levels of resistance achieved through this approach.
Several groups

have shown that this approach is also effective for protecting tobacco
against TSWV

(7,18,20). During the last two years we have made significant progress
towards applying

this technique to floral crops.
Ornamental crops pose a challenge for the development of resistance through

genetic engineering both because of the diversity of species and cultivars
available as well as

a lack of extensive studies on tissue culture protocols. At an international
symposium on in

vitro culture of horticultural crops held in Baltimore in June, 1992, only
15 of the 83

presentations given dealt specifically with ornamental crops. More troubling,
perhaps, is the

lack of information provided in many of these presentations on regeneration
and

transformation protocols. In order for both the public and private sector
to make advances in

the improvement of ornamental crops through genetic engineering, there
needs to be an

information base from which to proceed. In all crops that have been studied,
both shoot

regeneration and transformation are highly cultivar-specific. We have chosen
to use

chrysanthemum as a model system for floral crop transformation since it
has been the most

extensively studied.
In our previous work we successfully developed shoot regeneration protocols
for three

cultivars of chrysanthemum (27) using modifications of a protocol provided
by Dr. R.

Trigiano (University of Tennessee). Initial efforts for developing transformation
protocols

centered on the identification of strains of Agrobacterium tumefaciens
which were virulent on

the three chrysanthemum cultivars. A successful transformation protocol
was then developed

for the cultivar ‘Iridon’ using a disarmed vector strain carrying a vector
plasmid with two

marker genes, one for kanamycin resistance and the other for B- glucuronidase
(GUS)

activity. Kanamycin-resistant plants were regenerated, rooted, and transferred
to soil in the

greenhouse. Plants were also shown to have high levels of GUS activity,
thus confirming

stable transformation.
During the last year we have refined and modified our transformation procedure
and

have determined that it is effective for moving genes into both pot and
cut flower mums.

We
have also constructed several versions of the TSWV nucleocapsid gene and
have tested

these constructs for their ability to impart resistance to TSWV in tobacco
(20). Based on the

results of those experiments, we chose the most effective constructs and
successfully

transformed them into chrysanthemum. Using both mechanical and thrips inoculation
with a

highly virulent chrysanthemum isolate of TSWV, we have identified plants
which show a

high level of virus resistance.
Due to the importance of the N gene for resistance as well as its importance
as a

determinant of classifying Tospoviruses, we are also currently conducting
detailed

investigations of the variability of the N gene between the different isolates
of Tospoviruses.

We have developed a panel of monoclonal antibodies which provide a measure
of relatedness

of the various Tospoviruses (10). One of these monoclonal antibodies reacts
to both TSWV

and INSV. Thus, this monoclonal antibody has potential for simplifying
current serological

testing procedures as well as providing an increased ability to detect
unrecognized viruses.

OBJECTIVES
AND ANTICIPATED BENEFITS
The overall objective of our current research is to use chrysanthemum as
a model

system to investigate the efficacy of transforming plants with the virus
nucleocapsid gene for

the development of TSWV and INSV-resistant ornamental crops. We have three
specific

objectives for the coming year:
1) Determine the efficacy of our transformation protocol on a broader range
of

chrysanthemum cultivars. If the protocol has broad applicability, it will
pave the way for

using genetic engineering techniques for improvement of diverse cultivars.
Such efforts are

not limited to introduction of virus resistance. Given the rapid progress
in identifying and

isolating genes from plants and other organisms, such efforts will also
allow for introduction

of genes for other horticultural traits such as flower color or delayed
senescence.
2) Identify highly resistant plants of both pot and cut flower mums, and
test them for

resistance against diverse virus isolates. Successful deployment of the
resistant transgenic

plants will require that they express resistance to a broad range of virus
isolates found in

commercial settings. Some of the different constructions of the nucleocapsid
gene used to

transform the plants were designed to provide broad-spectrum resistance.
All transformed

plants will be tested against a diversity of isolates. Those with the broadest
resistance will

be used for objective 3.
3) To begin to test the transformed, resistant plants for both resistance
and quality

traits in a commercial setting. The ultimate test of any variety is its
performance in a

commercial setting. Tests are needed both to measure the stability and
efficacy of the

resistance to virus isolates and inoculum load present under commercial
conditions. It is also

necessary to determine that critical quality characteristics of the variety
have not been altered

by the genetic engineering procedure.
Benefits. TSWV & INSV are a widely respected problem in floral crop
production

(11, 12, 19). Stringent, laborious controls aimed at removing infected
planting material from

propagation and production areas as well as preventative measures to control
thrips (the

primary means of spread of this virus in floral crops) are both required.
Both measures are

expensive in terms of materials and labor for implementation. Using chrysanthemum
as a

model, our goal is to develop resistant varieties, thus reducing the reliance
of the floral

industry on these expensive measures as well as reduce sources of virus
inoculum. Success

with chrysanthemum will provide an excellent test case for extending this
technology to other

highly susceptible floral crop species such as gloxinia and impatiens.

MATERLALS AND METHODS
1. Chrysanthemum cultivar transformation. Using modifications of the methods

recently reported by our lab (27), we have been successful in transforming
both a pot and cut

flower mum variety. For this work we conducted extensive studies on variables
such as

explant source, appropriate strains of Agrobacterium (2, 27), hormones
and hormone

inhibitors (3), and temperature and light intensity and wavelength (24).
Regeneration and

transformation protocols are often highly cultivar-specific, however, our
success with two

genetically different varieties suggest that our protocol may have broad
applicability. If so,

such a protocol will allow for the efficient use of genetic engineering
technology for

chrysanthemum improvement. We will work with representatives of the floral
industry to

identify key varieties which differ in important horticultural traits.
Explant tissue from these

varieties will be tested for transformation ability. Transformed plants
will be selected on

kanamycin-containing medium, and the presence of the TSWV gene confirmed
by Southern

blot analysis and/or polymerase chain reaction (PCR). For constructs which
produce

proteins, levels of protein expression will be assayed by serological techniques
(Western

blots). Plants vegetatively propagated from the original transformants
will be tested for

stability of the gene, virus resistance and varietal characteristics. Success
in this work will

provide information needed to determine the practicality of genetic engineering
for

chrysanthemum improvement.
2) Test resistant plants against diverse virus isolates. Resistance to
the viruses in the

transformed plants will be conducted by both thrips and mechanical inoculation
techniques

with a representative sample of virus isolates. Transmission efficiency
using mechanical

inoculation is somewhat low for chrysanthemum as compared to other crops,
and we are

investigating strategies to improve the inoculation efficiency. Both temperature
and virus

isolate have proved critical in successful mechanical inoculation. Inoculum
is produced from

plants of Nicotiana benthamiana systemically infected with virus. Infected
leaves are ground

in buffer, and plants are inoculated by rubbing the inoculum onto leaves
which have been

dusted with carborundum to wound the leaves. Thrips inoculation is conducted
by rearing

thrips on chrysanthemum plants infected with the virus. Young cuttings
of transformed

plants are placed around the inoculum plants for five weeks, after which
they are sprayed to

eradicate the thrips and observed for the development of symptoms. This
procedure has

proved to be highly effective in screening of our transgenic. plants. Quantitative

determination of virus resistance is determined by measuring virus titre
in the upper leaves of

the plant using a serological assay (ELISA).
3) Test transformed plants in a commercial setting. Plants found to carry
high

levels of resistance to a broad range of isolates will need to be tested
in a commercial

setting. We will work with Dr. R. K. Jones, our department’s extension
specialist on

ornamental crops, and with representatives from industry to identify settings
and experiments

for evaluating both virus resistance and possible alterations in horticultural
traits of our most

promising transgenic plants. These studies will require approval from State
and Federal

regulatory agencies, as such a test involves release of genetically engineered
plants.

Discussions with Federal officials have indicated that a full application
will be necessary, as

the nucleocapsid gene of these viruses is not included in the exemptions
granted to

experiments conducted with transgenic plants which express “conventional”
virus capsid

protein genes. We have an excellent working relationship with regulatory
officials in North

Carolina, and anticipate no problems in putting together the necessary
documentation.
Facilities and Equipment.
We currently have all of the major equipment items, growth rooms and transfer
hoods

for the tissue culture and plant transformation research. In addition we
have adequate

greenhouse space. We also have all of the major items needed for the virology
portion of

the research. The budget requests funds for the salary of a technician
needed to carry out

the work. Also a budget for expendable supplies is included.
LITERATURE CITED
1. Beachy, R. N., Loesch-Fries, S., and Turner,
N. E. 1990. Ann. Rev.

Phytopathology 28:451-474.

2. Bush, A. L. and S. G. Pueppke. 1991. Physiol.
Molec. Plant Pathol. 39:309-323.

3. Chraibi, K. M., A. Latche, J. P. Roustan, and
J. Fallot. 1991. Plant Cell Rept.

10:204-207.

4. de Avila, A. C., de Haan, P., Kitajima, E. W.,
Kormelink, R., Resende, R. dde O.,

Goldbach, R., Peters, D. 1992. J. Phytopathology 134:133-151.

5. de Avila, A. C., Huguenot, C., Resende, R.,
de O., KitaJima, E. W., Goldbach, R.

W., and Peters, D. 1990. J. Gen. Virol. 71:2801-2807.

6. de Haan, P., Wagemakers, L., Peters, D., and
Goldbach, R. 1990. J. Gen. Virol.

71:1001-1007.

7. Gielen, J. J. L., deHaan, P., Kool, A. J., Peters,
D., van Grinsven, M. Q. J. M.,

and Goldbach, R. W. 1991. Bio/Technology 9:1363-1367.

8. German, T., Ullman, D., and Moyer, J. W. 1992.
Ann. Rev. Phytopathology

30:315-348.

9. Golelmboski, D. B., Lomonossoff, G. P., and
Zaitlin, M. 1990. Proc. Nad. Acad.

Sci. USA 87:63311-6315.

10. Hall, J. M. and Moyer, J. W. 1992. Annual Meeting
of American Society of

Virology. July 1992.

11. Jones, R. K. and Moyer, J. W. 1986. N. C. Flower
Growers Bulletin 30:11-13.

12.
Jones, R. K. and Moyer, J. W. 1987. North Carolina Flower Growers Bulletin

31:1-2.

13.
Kormelink, R., Kitajima, E. W., deHaan, P., Zuidema, D., Peters, D., Goldbach,
R.

1991. Virology.181:459-468.

14.
Law, M. D. and Moyer, J. W. 1989. Phytopathology 79:1157.

15.
Law, M. D. and Moyer, J. W. 1990. J. Gen. Virology 71:933-938.

16.
Law, M. D., Speck, J., and Moyer, J. W. 1991. J. Gen. Virology 72:2597-2601.

17.
Law, M. D., Speck, J. and Moyer J. W. 1992. Virology 188:732-741.

18.
MacKenzie, D. J. and Ellis, P. J. 1992. Molec. Plant-Microbe Interact.
5:34-40.

19.
Moyer, J. W. and Jones, R. K. 1991. Ball Redbook. Ball Publications.

20.
Perez, R. S. M. Geske, J. Speck, P. Reece, J. W. Moyer, and M. E. Daub.
1993.

Phytopathology 83:1355 (Abstract).

21.
Powell-Abel, P., Nelson, R. S., De, B., Noffman, N., Rogers, S. G., Fraley,
R. T.,

and Beachy, R. N. 1986. Science 232:738-743.

22-
Sanford, J. C. and Johnston, S. A. 1985. J. Theor. Biol. 113:395-405.

23.
Sherman, J. M., L. A. Urban, S. M. Geske, J. Speck, J. W. Moyer, and M.
E.

Daub. 1993. Phytopathology 83: 1355 (Abstract).

24.
Stasinopoulos, T. C., and Hangarter, R. P. 1990. Plant Physiol. 93:1365-1369.

25.
Torres, K. C. 1983. Tissue Culture Techniques for Horticultural Crops,
Chpt 6.

Van Nostrand Reinhold, NY.

26.
Urban, L. A. Huang, P.-Y. and Moyer, J. W. 1991. Phytopathology 81:525-529.

27.
Urban, L. A., J. M. Sherman, J. W. Moyer, and M. E. Daub. 1994. Plant Science

98: 69-79.

28.
Urban, L. A., J. Speck, J. W. Moyer, and M. E. Daub. 1992. Phytopathology

82:1147 (Abstract).

29.
Wang, M., and Gonsalves, D. 1990. Plant Disease 74:154-158.

BUDGET REQUEST

Technical Support
$26,000
Benefits (24.1%)
$ 6,266
Total Salaries
$32,266
Supplies
$ 5,000
TOTAL REQUESTED
$37,266

PROJECT LEADER QUALIFICATIONS
James W. Moyer is a professor of plant pathology at North Carolina State
University.

He received his Ph. D in plant pathology at The Pennsylvania State University
in 1975 and

did post-doctoral research at the University of California at Davis prior
to coming to NCSU.

He has had 18 years experience in working with viruses of vegetatively
propagated crops and

seven years experience investigating tomato spotted wilt-like viruses (Tospoviruses).

Members of his research group were responsible for first identifying INSV,
the predominant

virus problem in floral crops, and for providing antisera to the industry
for diagnosis and

clean-stock programs. His research includes both applied and basic aspects
of plant virology.
Margaret E. Daub is a professor of plant pathology at North Carolina State

University. She received a Ph.D. in plant pathology from the University
of Wisconsin at

Madison and did postdoctoral work in plant tissue culture at Michigan State
University. She

has had 15 years experience working with all aspects of plant in vitro
culture and

transformation, and has written several review articles on this subject.
Her research has

included in vitro selections of callus and protoplast cultures for phytotoxin
resistance, somatic

hybridization for transfer of resistance genes in plants, somaclonal variation
for improved

disease resistance, and plant transformation for pathogen gene expression.